COVID-19 causes critical health damage. The effects of the virus on mental and physical health can potentially lead to death. The COVID-19 is capable of causing Severe Acute Respiratory Syndrome. Furthermore, it transforms from the common cold to Middle East Respiratory Syndrome. The lungs of the patient are severely damaged.
However, the impact of the virus on the heart of the patient can have lethal outcomes. Furthermore, to explore the impact, researchers conducted research. Moreover, a three-part journal series, Journal of the American College of Cardiology, published the study.
Heart Issues In COVID-19 Patients
Initially, about the pandemic coronavirus, all we know was that it causes respiratory infection. However, over time, the effect of the virus on the heart becomes clearer. The second most damaged organ after lungs due to COVID-19 is the heart. Twenty-five percent of the COVID-19 patients develop a myocardial injury. Moreover, they are likely to suffer from wounded myocardial tissues and weakened heart muscles.
Furthermore, thromboembolic disease or blood clots and arrhythmias develop in a recognizable number of COVID-19 patients. In thromboembolic disorders, blood clots form in blood vessels. The clots block the flow of blood, especially in arteries. The results can be fatal, depending upon the type of blocked-artery. Moreover, in the U.S., thromboembolic disorders are a common cause of death in pregnant women.
Impact Of The Virus On Pre-existing Heart Problems
Dr. Aeshita Dwivedi is a cardiologist at Lenox Hill Hospital in New York City. According to Dwivedi, research has determined that the virus affects multiple organs of the patient, including the heart. The author of the study reports that the degree of heart damage in COVID-19 patients depends upon the pre-existing condition of their heart.
Furthermore, the author of the study recommends heart patients to make heart-healthy lifestyle changes. Moreover, to lower their risk of developing complications if infected with the COVID-19.
Pre-existing heart problems include hypertension, obesity, high cholesterol, and high blood sugar levels. Such patients are at a greater risk of developing lethal complications with COVID-19.
Action Of COVID-19 On The Cardiovascular System
There are two mechanisms through which the virus affects the heart. Firstly the virus causes an infection that causes inflammation across the whole body of the infected-patient. Thus, resulting in hindrance for the heart to perform its function. According to Dwivedi, the coronavirus infection causes the heart muscles to become feeble and forms abnormalities in heart rhythm. Furthermore, the formation of a clot in blood vessels is another possibility.
Secondly, COVID-19 invades Angiotensin-Converting Enzyme 2 receptors and causes direct viral harm. ACE2 receptors are present within myocardial tissues and are responsible for blood flow and blood pressure maintenance.
According to Dr. Benjamin J. Hirsh, the virus is capable of affecting heart muscles. Furthermore, through inflammation or by direct-invasion, the virus can cause damage to heart muscles and ultimately lead to heart failure.
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Is The Damage Reversible, Caused By The Virus?
The risk of heart damage in COVID-19 patients is high if the patient has pre-existing heart problems. However, according to research, the damage also depends upon the amount of viral inoculum and the individual’s immune response.
Research is under process to discover how long the damage of COVID-19 infection will last. However, early evidence shows that heart damage symptoms in coronavirus patients last for months.
Furthermore, 87% of the patients showed at least one persistent symptom. These symptoms include chest pain, joint pain, and fatigue. According to Dwivedi, the complications are long-lasting, associated with COVID-19 and pre-existing heart disease.
Furthermore, an individual needs to improve their lifestyle and eating habits and follow precautionary measures. Moreover, avoid unhealthy habits to prevent the development of complications associated with the disease.